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Full article "Abstract: High dose glucocorticoid (GC) administration impairs the viability and function of osteoblasts, thus causing osteoporosis and osteonecrosis.Neuropeptide Y1 receptor (Y1 receptor) is expressed in bone tissues and cells, and regulates bone remodeling.Below is the scheme of the exon-intron sequence spanning from exon 20 to 23.The colored bars represent exons 20–23 of the gene, at both the genomic (I) and c DNA level (II and III).Full article "Figure 7 Docking interactions of dexibuprofen with receptor.

Osteoblast differentiation was evaluated by Alizarin Red S staining and osteogenic marker gene expressions.

Taken together, Y1 receptor regulates Dex-induced inhibition of osteoblast differentiation in murine MC3T3-E1 cells via ERK signaling.

This study provides a novel role of Y1 receptor in the process of GC-induced suppression in osteoblast survival and differentiation.

The COX-2 protein chain is highlighted in light grey color in wire format.

The dark grey color showed the ligand structure while the embedded red colors showed oxygen moiety.

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